CBD uses include: visual focus, mental clarity, reaction time improved, hold breath longer, ulcerative colitis, calms down inflammation and cramping, anti-tumor necrosis factor.
CBD works at the mitochondria level in the cell via oxidative phosphorylation which increases energy.
In regards to cancer, CBD protects normal cells while signaling cell death in abnormal cells. It may prevent metastasis and also paralyzes cells at the mitochondria level. A noted effective dose for cancer patients is 90 mg.
In regard to sleep, it reduces the amount of REM anxiety sleep and increases deep sleep which is noted as stage 4 sleep. It is important to dose CBD 1 to 2 hours before sleep as it can actually activate the brain.
In regard to auto immune system attack:
CBD is effective and rheumatoid arthritis, irritable bowel syndrome, lupus, thyroid disorder, fibromyalgia, multiple sclerosis, skin psoriasis and eczema and chronic infection.
In regard to brain inflammation:
CBD is effective to reduce brain fog, migraine, epilepsy, anxiety, PTSD, ADHD, ADD, autism and insomnia. Research indicates an anandamide levels are higher in the brain of ADHD subjects. Anandamide binds to the CB1 receptor through the G protein coupling system. CBD has an indirect effect on the CB1 receptor by stopping the enzymatic breakdown of Anandamide. CBD helps to mediate the release of cytokines from the immune cells in a manner that helps to reduce inflammation and pain. Cannabinoid receptors (CB1 and CB2) are found in high concentrations in areas of the brain that are malfunctioning in ADHD, such as the amygdala and hippocampus.
In regard to Patients with endometriosis: it is determined these patients often suffer from diffuse and poorly localized severe pain. The current pain management strategies include medical and hormonal therapy, as well as surgery. Medical management of pain is often insufficient and is associated with high rate of recurrence. Better pain management is therefore of urgent need.
The endocannabinoid system (ECS) has recently emerged as a relevant pharmacological target for the management of endometriosis-related pain. Mostly, endometriosis-associated pain can be targeted by modulation of the ECS by modulation and signaling of anti-inflammatory properties.